By James J. Reidy MD
Discusses the constitution and serve as of the cornea and exterior eye and stories suitable exam options. Covers infectious and ocular floor illnesses, issues and surgical procedure of the ocular floor, immune-mediated and neoplastic problems, congenital anomalies and degenerations. A lately up to date bankruptcy at the genetics of corneal dystrophies displays the recent IC3D category. Discusses poisonous and annoying accidents and corneal transplantation. includes many new colour photos. lately revised 2010 2011.
Read or Download 2011-2012 Basic and Clinical Science Course, Section 8: External Disease and Cornea (Basic & Clinical Science Course) PDF
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Additional info for 2011-2012 Basic and Clinical Science Course, Section 8: External Disease and Cornea (Basic & Clinical Science Course)
Brandt JD. Corneal thickness in glaucoma screening, diagnosis, and management. 2004;15(2}:85-89 . Doughty M], Zaman ML. Hu man corneal thickness and its impact on in traocular pressure measures: a review and meta -analysis approach. Surv Ophthalmol. 2000;44(5):367-408. Esthesiometr Esthesiometry is the measurement of corneal sensation, which is a function of the ophth almic branch of cranial nerve V. Its primary use is in the evaluation of neurotrophic keratopathy. In most clinical circumstances) reduced corneal sensitivity can be diagnosed qualitatively without special instruments, but quanti tative esthesiometry is useful in unusual cases and for research.
Interpretation of Ocular Cytology Microscopic examination of material collected from the ocular surface can reveal ceils, cell ular elements, and microorganisms that can be helpful in diagnostic evaluation; such examination is perhaps best carried out in conjunction with a laboratory experienced in these evaluations. Dry-Eye Syndrome The term dry-eye syndrome has been defined as "a multifactorial disease of the tears and ocular surface that results in symptoms of d iscomfort, visual disturbance, and tear-ftlm instability with potential damage to the ocular surface.
The most common cause is inflammatory lacrimal damage, which is seen in autoimmune disorders such as Sjogren syndrome and also in non-Sjogren syndrome dry eye (NSSDE). Inflammation causes both tissue destruction and a potentially reversible neurosecretory block. A receptor block may also be caused by circulating antibodies to the M3 receptor. Inflammation is favored by low tissue androgen levels. Tear delive ry may be obstructed by cicatricial conjunctival scarrin g or reduced by a loss of sensory reflex drive to the lacrimal gland from the ocular surface.
2011-2012 Basic and Clinical Science Course, Section 8: External Disease and Cornea (Basic & Clinical Science Course) by James J. Reidy MD